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thelostreceptor last won the day on May 27 2019

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About thelostreceptor

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  1. Neuronal/axonal damage is tied with the forming of glial scars that inhibit regeneration of tissue. Glial scars can be seen as a persistent deficit, and they create a non-permissive barrier that prevents axons growing through the area and re-connecting with targets. Unlike regular scar tissue, a glial scar does not really resolve and likely persists over one's lifetime. Glial scars have been known for long time as a major roadblock to regeneration and functional recovery, and there are many companies trying to solve this problem. There's loads of literature available, I found these wiki p
  2. Thanks, the studies you've sent are very interesting, and it's good to see counterarguments against an atrophic nature of this disorder. I don't know your background, but we're not all scientists and we could be biased by our beliefs in the cause, thus discussion should be sparked and in the best case we work towards a consensus. This is a good thing. But if neuronal loss is not the trigger, then there might even be less hope for a cure in the future, because the mechanisms could involve very weird alterations in neurochemistry that have to be specifically researched for this disorde
  3. Thought I'd post this in case you didn't notice it, it's a recent study (Apr 2020) about visual snow. https://pubmed.ncbi.nlm.nih.gov/32211752/ Structural and functional footprint of visual snow syndrome Patients with visual snow syndrome suffer from a continuous pan-field visual disturbance, additional visual symptoms, tinnitus, and non-perceptional symptoms. The pathophysiology of visual symptoms might involve dysfunctional visual cortex. So far, the extra-visual system has not been investigated. We aimed at identifying structural and functional correlates for visual and
  4. Thanks for the extensive research, I agree that the toxicity of drugs is questionable in science in general, there are studies conflicting with each other. I think your conclusion for mitochondrial dysfunction is reasonable, it just feels like something is missing. Because the mitochondria could be changed and altered in response to environmental factors, I don't know them to be the core of the disease. But yeah the reversal of certain migraines with riboflavin/vitamin b2 is very interesting. Is HPPD an umbrella term? Are the underlying causes of cases distinct from each other, or
  5. I've been reading about neuronal hyperexcitability, I'm keen that HPPD could be related in some way to this. That would mean HPPD is caused by a brain plasticity mechanism called 'homeostatic plasticity' which upregulates a neural networks output beyond their regular range, causing persistent hyperexcitability. Also there is a connection to acquired epilepsy as well as tinnitus, which are driven by excessive excitability. Tinnitus can be induced by too much noise exposure but also through brain injury which seems to be closer related to HPPD. The homeostatic plasticity acts to stabil
  6. Thanks @Jay1 for fixing the forum, this site and the information on it has been invaluable for me, also the new design is stunning! There's just another message on the homepage from David starting with "I AM SO SORRY. I did not realize the gallery I used to upload m....", perhaps we could get rid of that as well because it's been there for a very long time.
  7. I would too lean towards a hypothesis of some sort of 'damage' or change as my case was not caused by classic hallucinogens but after the incident of combining two serotonergic drugs (one of them being MDMA). I appreciate and respect @dasitmane for exploring all the avenues but for myself, I prefer to focus on the brain. I believe this condition should be fully reversible at some point in the future.
  8. Thanks yosoydiego, just wanted to let you and people reading this know in case you weren't aware of it
  9. Thank you dasitmane for coming back on this forum, I was following your first thread with great pleasure. I hope that we can make some progress in our knowledge and that people will join in and share their findings. @yosoydiego About the Reddit thread, the connection with HPPD and Lyme was reported by a user who is actively promoting the use of chlorine dioxide to (falsely) cure HPPD. It is very unhealthy to practice this 'cure' and will not aid your mind and body in any positive way, so take that in mind when reading posts made by that user (HPPDandstuff).
  10. For me, alcohol doesn't seem to have a negative effect on my visuals, it even seems to reduce visual snow by just a little temporary It is the additional use of cigarettes that temporary (1 - 2 days) gives a notable increase in the intensity of visual snow and causes closed-eye light flashes
  11. subscribed Thank you David for all your dedication throughout the years you've been suffering from this, without these forums here I would have never had a new outlook on life
  12. Might also help with visual snow. Here's an anecdotal report of someone who had her visual disturbances improved by daily intranasal insulin intake. The downside is that she reported it returned full blown upon cessation of the insulin Check the full thread here: https://www.longecity.org/forum/topic/89360-intranasal-insulin-dihexa-log/page-2#entry796569
  13. By anecdote, I have a friend who got visual snow from a stimulant. He was 19 when it happened and the symptoms subsided after a month. Unfortunately for me, my own visual snow is still here after two years. Note that we haven't used together. If you're still in adolescence it would make sense that your brain could eventually fix or adapt to the changes. Reading that you're 16 and got symptoms from weed, I suspect you're likely to have a positive outcome for your situation. Take care of yourself and stay away from weed and other drugs
  14. Thanks for posting your findings on these forums. I've actually read your story somewhere else before, and I'm wondering: what was the cause of your VS? Did you experience other symptoms similar or related to HPPD?
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