Fawkinchit

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5-HT2A is expressed widely throughout the central nervous system (CNS). It is expressed near most of the serotoninergic terminal rich areas, includingneocortex (mainly prefrontal , parietal , andsomatosensory cortex ) and the olfactory tubercle . Especially high concentrations of this receptor on the apical dendrites of pyramidal cells in layer V of the cortex may modulate cognitive processes,[8] [9] [10] by enhancing glutamate release followed by a complex range of interactions with the 5-HT1A ,[11] GABAA ,[12] adenosine A1 ,[13] AMPA ,[14] mGluR2/3 ,[15] mGlu5 ,[16] andOX2 receptors.[17] [18] In the rat cerebellum, the protein has also been found in the Golgi cells of the granular layer ,[19] and in the Purkinje cells .[20] [21]

Ah right, an overexcitation of specific neuron.

Mg could you elaborate on what you're posting

Retigabine is just a potassium channel opener. Not sure what the significance of this is?

You got that from the HPPD or already had it? Edit nvm guess its caused by the antipsychotics

Mg did it help your anxiety at all?

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This is a good point as well. But maybe the damage is somewhere else that just damage to the nuerons, maybe it has to do with damage related to the synapses etc...

Thats a good question.

Thats awesome please do i would like to see that. That would explain why some people are getting these symptoms from SSRIs and why syrian rue become hallucinogenic

Would you like to be more descriptive?

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Wow, maybe thats it! maybe 5ht2a receptors become permanitly down regulated! hence the need for an inverse agonist to reverse the effect. Possible, but still a shot in the dark.

Hey guys i'm still here but don't have internet or much time due to work. Hope - you don't HAVE to have a phd, right there you already failed in curing this, they have a brain, you have a brain, what's the difference? They've spent more time researching. As for now the problem is somewhere in the CNS, we need to research everything about the CNS

Its just a beta blocker, it MAY treat symptoms. 5htp2a receptor Inverse agonists

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What? Im pretty sure youre reading my posts wrong...

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Please provide the evidence of claims, I have seen absolutely no usage of inverse 5HT2A agonists for the treatment of hppd. Nor have I seen that gene mutation is negligible.