Whatever

Risperidone is a No No for hppd. In the farmacology section of Risperidone is actually a inverse agonist of the 5ht2a receptor ( the one that we think hppd is affected ) That means that it actually bind to the receptor without producing any effect. But to some already sensitive 5ht2a receptors this is actually wrong,because in such scenario activation seems to happen. I know because i was given risperidone and symptoms worsened. Stay away from that stuff. Is such a cheap and strong medication that's why they give it so much but it actually is dirt, there are other way better than risperidone. Doctors get sanctioned for prescribing costly medications. Anyways, hope you get well. Stay safe!

This is are some questions for a survey. I want to know how many of you have a history of illness? Did your parents have a story of illness? Specially of lyme disease, or aids or hiv or inmuno-supression, infections of any kind, that kind of stuff. What diagnosis have you ever had throught your medical history. Also I want to know how many of you have ever gotten bitten by a tick? Even if it was many many years ago. At whatever point in your life. Ever bitten by any other bug? How long ago have you been bitten? Are there plenty of mosquitoes in the area you live? Do they bite you quite often? Also, have you been in contact with cat feces? Have you ever been scratched by a cat, or dog , or any other animal? Have you ever tested positive for any kind of pathogen? Be a virus, bacteria, parasite or some other. Have you any autoinmune disease? Lupus? Multiple sclerosis? Parkinsonism? Gut issues? Alergies? Skin isses? Anything you might have feel free to share it, it will be helpful. How many of you got a rash like in the image at some point in your life? It is a rash that starts like a tiny red dot and with every day passed it gets bigger and bigger. ( If you are wondering, is a lyme disease rash, also known as erythema migrans, it shows after borrelia bacteria infection ) It goes away on itself after some time. Might eventually be missdiagnosed as fungal skin infection. Have you ever had hppd like manifestations before having hppd? Did your hppd start right before the last psychdelic use, or it was weeks after the cesation of use? Thanks! All this questions will be completelly anonymous. If you are not willing to share them here publicly, you can hit me answering them on direct message. This is for gathering anectodal data, this is nothing oficial, just out of curiosity and of individual research. Take care.

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Quoting the wikipedia: Human polyomavirus 2 can cross the blood–brain barrier into the central nervous system, where it infects oligodendrocytes and astrocytes, possibly through the 5-HT2A serotonin receptor.[14]Human polyomavirus 2 DNA can be detected in both non-PML affected and PML-affected (see below) brain tissue.[15] So this virus can be in healthy human beings without having to provoke PML ( the disease I mean ) From webmed.com (https://www.webmd.com/brain/jc-virus#1) Experts don't know how it's spread, but it's thought that many people pick it up as kids through food or water that has the virus in it. It settles in your urinary tract, bone marrow, tonsils, or brain. It can stay there for years, and most people never know they have it. From https://www.healthline.com/health/multiple-sclerosis/jc-virus-risks-for-ms-patients : The John Cunningham virus, known more commonly as the JC virus, is a very common virus in the United States. According to the World Journal of Neurosciences, between 70 and 90 percent of people in the world have the virus. The average person carrying the JC virus will never know and is unlikely to experience any side effects. So most people have it right? Having it doesnt mean you are getting diseased, as with every microorganism. So if you pay close attention, to this video You can notice how a virus when attaches to a receptor activates it. Just keep in mind that LSD or Psilocin is "the key" as well, and the lock is your neuronal receptor. As well as the JC virus that can lock in the same receptors that psychedelics do as well. The 5ht2a receptor, a serotonin receptor. Since in a sinapse there is more than one serotonin 5ht2a receptor, couldnt it be that the virus is locking to the receptors as they open up since the viral shape is round and is sticking to one and to another several times? I mean if a virus locks into a normal cell, it would enter the cell. But in neurons, when receptors are activated , they dont let in anything inside, that just triggers an electrical impilse to the nearest neuron, right? Depolarization of the neuron membrane is called. Watch david nichols videos about neuroscience and you will understand this better. Neurons are specialized cells and they get fed and all their essential functions are done by their nearby glial cells. So they dont need to le inside them anything, that is already done by the cells around them. So, we know that if serotonin is activating a receptor ( wich is usually the thing that activates it ) that receptor is filled with serotonin and wont let in a virus or substance. And after the serotonin is expelled, that receptor is closed for a time until is open again so can accept serotonin again. Where I'm trying to get here, is that when something is activating the receptor ( lsd, psilocin, serotonin and alikes ) , the triggering neuron wont let more serotonin inside the synaptic space, because the triggered neuron doesnt detect that there is a serotonin depletion in the synaptic space. So serotonin in the synaptic space is depleted. So when there is no serotonin, anything that can enter those 5ht2a recetprs is going to enter into them. Since serotonin has more afinity to them, serotonin has prefecernce over the JC virus to enter the receptor. But since there is no serotonin because the psychedelic has made it to get depleted, the JC virus enters the receptor instead, activating the neuron again, and creating a vicious cycle, where serotonin is constantly depleted and there is nothing that can expell the virus away, and it will keep activating the 5ht2a receptors around. Thus creating the symptomatology of perma tripping known as HPPD or visual snow. Makes sense? To me makes 100% sense. I dont know why nobody is talking about this. Like there is a viral agent that can activate the very same receptors as psychedelics do, and nobody is talking about it. And viruses are the most widespread disease causing agents and still no one has ever thought about it? I just cant believe it. Maybe is not the drugs, maybe is the virus. Since, there are many people that get the hppd weeks later after having the psychedelic experience and trying weed. And they then escalate slowly but steady. Just like an infection And by the way, regarding the visual snow syndrome, if the viral count of this virus gets real big for whatever reason, they can bind directly to the 5ht2a receptor without the need of a psychedelic. It's called selevtive affinty. Just my 2 cents.